期刊
NATURE NEUROSCIENCE
卷 18, 期 4, 页码 477-478出版社
NATURE PUBLISHING GROUP
DOI: 10.1038/nn.3986
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资金
- NINDS NIH HHS [R01 NS037853] Funding Source: Medline
Reductions in brain glucose metabolism have long been associated with Alzheimer's disease. A study now demonstrates that the endothelial glucose transporter GLUT1 is vital for maintaining brain energy metabolism and vascular clearance of amyloid-beta.
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