相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。New IBD genetics: common pathways with other diseases
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Interaction of the major inflammatory bowel disease susceptibility alleles in Crohn's disease patients
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Epistasis Between Toll-Like Receptor-9 Polymorphisms and Variants in NOD2 and IL23R Modulates Susceptibility to Crohn's Disease
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Investigation of Crohn's Disease Risk Loci in Ulcerative Colitis Further Defines Their Molecular Relationship
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Strong Evidence of a Combination Polymorphism of the Tyrosine Kinase 2 Gene and the Signal Transducer and Activator of Transcription 3 Gene as a DNA-Based Biomarker for Susceptibility to Crohn's Disease in the Japanese Population
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Common variants at five new loci associated with early-onset inflammatory bowel disease
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Replication of signals from recent studies of Crohn's disease identifies previously unknown disease loci for ulcerative colitis
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Genetic determinants of ulcerative colitis include the ECM1 locus and five loci implicated in Crohn's disease
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Genome-wide association defines more than 30 distinct susceptibility loci for Crohn's disease
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NATURE GENETICS (2008)
The IL23 axis plays a key role in the pathogenesis of IBD
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IL23R variation determines susceptibility but not disease phenotype in inflammatory bowel disease
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Genome-wide association study identifies new susceptibility loci for Crohn disease and implicates autophagy in disease pathogenesis
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A genome-wide association scan of nonsynonymous SNPs identifies a susceptibility variant for Crohn disease in ATG16L1
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