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注意:仅列出部分参考文献,下载原文获取全部文献信息。Interleukin-18: A Mediator of Inflammation and Angiogenesis in Rheumatoid Arthritis
Michael V. Volin et al.
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IL-18 Inhibits TNF-α-Induced Osteoclastogenesis Possibly via a T Cell-Independent Mechanism in Synergy with IL-12 In Vivo
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Interleukin-18 induces angiogenic factors in rheumatoid arthritis synovial tissue fibroblasts via distinct signaling pathways
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Interleukin-18 induces the production of vascular endothelial growth factor (VEGF) in rheumatoid arthritis synovial fibroblasts via AP-1-dependent pathways
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Interleukin-18 induces serum amyloid A (SAA) protein production from rheumatoid synovial fibroblasts
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Interleukin (IL) 18 stimulates osteoclast formation through synovial T cells in rheumatoid arthritis:: comparison with IL1β and tumour necrosis factor α
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ANNALS OF THE RHEUMATIC DISEASES (2004)
Osteoclast differentiation and activation
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NATURE (2003)
Heterogeneity of response of rheumatoid synovium cell subsets to interleukin-18 in relation to differential interleukin-18 receptor expression
M Kawashima et al.
ARTHRITIS AND RHEUMATISM (2003)
Signal transduction pathways involved in rheumatoid arthritis synovial fibroblast interleukin-18-induced vascular cell adhesion molecule-1 expression
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Expression of interleukin-18 receptor in fibroblast-like synoviocytes
B Moller et al.
ARTHRITIS RESEARCH (2002)
Interleukin-18 induces rheumatoid arthritis synovial fibroblast CXC chemokine production through NFκB activation
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The effect of macrophage-colony stimulating factor and other humoral factors (interleukin-1,-3,-6, and-11, tumor necrosis factor-α, and granulocyte macrophage-colony stimulating factor) on human osteoclast formation from circulating cells
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BONE (2001)
Reduced incidence and severity of collagen-induced arthritis in mice lacking IL-18
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