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Dimorphic Motifs in D0 and D1+D2 Domains of Killer Cell Ig-Like Receptor 3DL1 Combine to Form Receptors with High, Moderate, and No Avidity for the Complex of a Peptide Derived from HIV and HLA-A*2402
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Synergistic polymorphism at two positions distal to the ligand-binding site makes KIR2DL2 a stronger receptor for HLA-C than KIR2DL3
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Expression of murine killer immunoglobulin-like receptor KIRL1 on CD1d-independent NK1.1+ T cells
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KIR2DS1-positive NK cells mediate alloresponse against the C2HLA-KIR ligand group in vitro
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A subpopulation of human peripheral blood NK cells that lacks inhibitory receptors for self-MHC is developmentally immature
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KIR2DL4 (CD158d) genotype influences expression and function in NK cells
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DNA methylation maintains allele-specific KTR gene expression in human natural killer cells
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The DO domain of KIR3D acts as a major histocompatibility complex class I binding enhancer
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SHP-1-and phosphotyrosine-independent inhibitory signaling by a killer cell Ig-like receptor cytoplasmic domain in human NK cells
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