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注意：仅列出部分参考文献，下载原文获取全部文献信息。Regulatory T cell expression of herpesvirus entry mediator suppresses the function of B and T lymphocyte attenuator-positive effector T cells
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Targeting the primary tumor to generate CTL for the effective eradication of spontaneous metastases
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Unexpected role of B and T lymphocyte attenuator in sustaining cell survival during chronic allostimulation
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The glycosaminoglycan-binding domain of decoy receptor 3 is essential for induction of monocyte adhesion
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Stimulating lymphotoxin β receptor on the dendritic cells is critical for their homeostasis and expansion
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The critical role of LIGHT in promoting intestinal inflammation and Crohn's disease
J Wang et al.
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B and T lymphocyte attenuator exhibits structural and expression polymorphisms and is highly induced in anergic CD4+ T cells
MA Hurchla et al.
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The role of herpesvirus entry mediator as a negative regulator of T cell-mediated responses
Y Wang et al.
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LIGHT is constitutively expressed on T and NK cells in the human gut and can be induced by CD2-mediated signaling
O Cohavy et al.
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A TNF family member LIGHT transduces costimulatory signals into human T cells
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Cutting edge:: Selective impairment of CD8+ T cell function in mice lacking the TNF superfamily member LIGHT
K Tamada et al.
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Visualization of lymphotoxin-β and lymphotoxin-β receptor expression in mouse embryos
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Constitutive expression of LIGHT on T cells leads to lymphocyte activation, inflammation, and tissue destruction
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Genomic characterization of LIGHT reveals linkage to an immune response locus on chromosome 19p13.3 and distinct isoforms generated by alternate splicing or proteolysis
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The TNF superfamily members LIGHT and CD154 (CD40 ligand) costimulate induction of dendritic cell maturation and elicit specific CTL activity
Y Morel et al.
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Signal via Lymphotoxin-βR on bone marrow stromal cells is required for an early checkpoint of NK cell development
Q Wu et al.
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Reciprocal expression of the TNF family receptor herpes virus entry mediator and its ligand LIGHT on activated T cells: LIGHT down-regulates its own receptor
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LIGHT, a TNF-like molecule, costimulates T cell proliferation and is required for dendritic cell-mediated allogeneic T cell response
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