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E. Kabashi et al.
JOURNAL OF NEUROCHEMISTRY (2008)
Depletion of 26S proteasomes in mouse brain neurons causes neurodegeneration and Lewy-like inclusions resembling human pale bodies
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Role of increased expression of the proteasome in the protective effects of sulforaphane against hydrogen peroxide-mediated cytotoxicity in murine neuroblastoma cells
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Disease-associated prion protein oligomers inhibit the 26S proteasome
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Pyrrolidine dithiocarbamate inhibits induction of immunoproteasome and decreases survival in a rat model of amyotrophic lateral sclerosis
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Insoluble mutant SOD1 is partly oligoubiquitinated in amyotrophic lateral sclerosis mice
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Molecular biology of amyotrophic lateral sclerosis: insights from genetics
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Proteasomal degradation of mutant superoxide dismutases linked to amyotrophic lateral sclerosis
L Di Noto et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2005)
Non-neuronal induction of immunoproteasome subunits in an ALS model: Possible mediation by cytokines
K Puttaparthi et al.
EXPERIMENTAL NEUROLOGY (2005)
Accumulation of human SOD1 and ubiquitinated deposits in the spinal cord of SOD1G93A mice during motor neuron disease progression correlates with a decrease of proteasome
C Cheroni et al.
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Neuronal dysfunction in a polyglutamine disease model occurs in the absence of ubiquitin-proteasome system impairment and inversely correlates with the degree of nuclear inclusion formation
AB Bowman et al.
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Evidence for systemic immune system alterations in sporadic amyotrophic lateral sclerosis (sALS)
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JOURNAL OF NEUROIMMUNOLOGY (2005)
Lessons from models of SOD1-linked familial ALS
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TRENDS IN MOLECULAR MEDICINE (2004)
CHIP promotes proteasomal degradation of familial ALS-linked mutant SOD1 by ubiquitinating Hsp/Hsc70
M Urushitani et al.
JOURNAL OF NEUROCHEMISTRY (2004)
Focal dysfunction of the proteasome: a pathogenic factor in a mouse model of amyotrophic lateral sclerosis
E Kabashi et al.
JOURNAL OF NEUROCHEMISTRY (2004)
A transgenic mouse model of the ubiquitin/proteasome system
K Lindsten et al.
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Proteasomal inhibition causes the formation of protein aggregates containing a wide range of proteins, including nitrated proteins
DH Hyun et al.
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Persistent activation of p38 mitogen-activated protein kinase in a mouse model of familial amyrotrophic lateral sclerosis correlates with disease progression
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Modulation of gene expression by cancer chemopreventive dithiolethiones through the Keap1-Nrf2 pathway - Identification of novel gene clusters for cell survival
MK Kwak et al.
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Analysis of the cytosolic proteome in a cell culture model of familial amyotrophic lateral sclerosis reveals alterations to the proteasome, antioxidant defenses, and nitric oxide synthetic pathways
S Allen et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2003)
Antioxidants enhance mammalian proteasome expression through the Keap1-Nrf2 signaling pathway
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Aggregate formation in the spinal cord of mutant SOD1 transgenic mice is reversible and mediated by proteasomes
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JOURNAL OF NEUROCHEMISTRY (2003)
Proteasomal inhibition by misfolded mutant superoxide dismutase 1 induces selective motor neuron death in familial amyotrophic lateral sclerosis
M Urushitani et al.
JOURNAL OF NEUROCHEMISTRY (2002)
Differentially expressed genes in sporadic amyotrophic lateral sclerosis spinal cords - screening by molecular indexing and subsequent cDNA microarray analysis
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The importance of the proteasome and subsequent proteolytic steps in the generation of antigenic peptides
AL Goldberg et al.
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The ubiquitin-proteasome proteolytic pathway: Destruction for the sake of construction
MH Glickman et al.
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Transgenic SOD1 G93A mice develop reduced GLT-1 in spinal cord without alterations in cerebrospinal fluid glutamate levels
C Bendotti et al.
JOURNAL OF NEUROCHEMISTRY (2001)
Cytokine upregulation in a murine model of familial amyotrophic lateral sclerosis
JL Elliott
MOLECULAR BRAIN RESEARCH (2001)
Early vacuolization and mitochondrial damage in motor neurons of FALS mice are not associated with apoptosis or with changes in cytochrome oxidase histochemical reactivity
C Bendotti et al.
JOURNAL OF THE NEUROLOGICAL SCIENCES (2001)
Medical progress: Amyotrophic lateral sclerosis.
LP Rowland et al.
NEW ENGLAND JOURNAL OF MEDICINE (2001)
Formation of high molecular weight complexes of mutant Cu,Zn-superoxide dismutase in a mouse model for familial amyotrophic lateral sclerosis
JA Johnston et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2000)
Tissue distribution of constitutive proteasomes, immunoproteasomes, and PA28 in rats
C Noda et al.
BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS (2000)