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Deletion of the SOCS3 gene in liver parenchymal cells promotes hepatitis-induced hepatocarcinogenesis
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Loss of SOCS3 in the liver promotes fibrosis by enhancing STAT3-mediated TGF-β1 production
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SOC1 inhibits HPV-E7-mediated transformation by inducing degradation of E7 protein
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STAT-1 interacts with p53 to enhance DNA damage-induced apoptosis
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SOCS1 is a suppressor of liver fibrosis and hepatitis-induced carcinogenesis
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SOCS1/JAB is a negative regulator of LPS-induced macrophage activation
I Kinjyo et al.
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SOCS-1/SSI-1-deficient NKT cells participate in severe hepatitis through dysregulated cross-talk inhibition of IFN-γ and IL-4 signaling in vivo
T Naka et al.
IMMUNITY (2001)
Inhibition of signal transducer and activator transcription factor 3 in rats with acute hepatic failure
Y Kamohara et al.
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Signals transducers and activators of transcription (STAT)-induced STAT inhibitor-1 (SSI-1)/suppressor of cytokine signaling-1 (SOCS-1) suppresses tumor necrosis factor α-induced cell death in fibroblasts
Y Morita et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2000)
Critical contribution of liver natural killer T cells to a murine model of hepatitis
K Takeda et al.
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