Journal
JOURNAL OF VIROLOGY
Volume 89, Issue 15, Pages 8082-8087Publisher
AMER SOC MICROBIOLOGY
DOI: 10.1128/JVI.01046-15
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Funding
- Battelle Memorial Institute
- U.S. National Institute of Allergy and Infectious Diseases [HHSN272200700016I]
- NIH, NIH-National Science Foundation Ecology of Infectious Disease program [TW009237, R01 AI077376]
- Office of Research Infrastructure Programs grant [P51OD011106]
- Intramural Research Program of the NIH, National Library of Medicine
- FOGARTY INTERNATIONAL CENTER [R01TW009237] Funding Source: NIH RePORTER
- NATIONAL INSTITUTE OF ALLERGY AND INFECTIOUS DISEASES [ZIAAI001025, ZIDAI009006, R01AI077376, R01AI098420] Funding Source: NIH RePORTER
- NATIONAL INSTITUTE OF GENERAL MEDICAL SCIENCES [T32GM081061, T32GM008692] Funding Source: NIH RePORTER
- OFFICE OF THE DIRECTOR, NATIONAL INSTITUTES OF HEALTH [P51OD011106] Funding Source: NIH RePORTER
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Simian hemorrhagic fever (SHF) is lethal for macaques. Based on clinical presentation and serological diagnosis, all reported SHF outbreaks were thought to be caused by different strains of the same virus, simian hemorrhagic fever virus (SHFV; Arteriviridae). Here we show that the SHF outbreaks in Sukhumi in 1964 and in Alamogordo in 1989 were caused not by SHFV but by two novel divergent arteriviruses. Our results indicate that multiple divergent simian arteriviruses can cause SHF.
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