Related references
Note: Only part of the references are listed.Meta-analysis of Published Studies Identified Eight Additional Common Susceptibility Loci for Crohn's Disease and Ulcerative Colitis
Junji Umeno et al.
INFLAMMATORY BOWEL DISEASES (2011)
Meta-analysis identifies 29 additional ulcerative colitis risk loci, increasing the number of confirmed associations to 47
Carl A. Anderson et al.
NATURE GENETICS (2011)
The kinase LRRK2 is a regulator of the transcription factor NFAT that modulates the severity of inflammatory bowel disease
Zhihua Liu et al.
NATURE IMMUNOLOGY (2011)
LRRK2 Is Involved in the IFN-γ Response and Host Response to Pathogens
Agnes Gardet et al.
JOURNAL OF IMMUNOLOGY (2010)
Genome-wide meta-analysis increases to 71 the number of confirmed Crohn's disease susceptibility loci
Andre Franke et al.
NATURE GENETICS (2010)
Loss of leucine-rich repeat kinase 2 causes impairment of protein degradation pathways, accumulation of α-synuclein, and apoptotic cell death in aged mice
Youren Tong et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2010)
LRRK2 regulates autophagic activity and localizes to specific membrane microdomains in a novel human genomic reporter cellular model
Javier Alegre-Abarrategui et al.
HUMAN MOLECULAR GENETICS (2009)
Genomewide Association Study of Leprosy
Fu-Ren Zhang et al.
NEW ENGLAND JOURNAL OF MEDICINE (2009)
Genome-wide association defines more than 30 distinct susceptibility loci for Crohn's disease
Jeffrey C. Barrett et al.
NATURE GENETICS (2008)
A strategy for probing the function of noncoding RNAs finds a repressor of NFAT
AT Willingham et al.
SCIENCE (2005)
Mutations in LRRK2 cause autosomal-dominant Parkinsonism with pleomorphic pathology
A Zimprich et al.
NEURON (2004)
NFAT signaling: Choreographing the social lives of cells
GR Crabtree et al.
CELL (2002)