Journal
EXPERIMENTAL PHYSIOLOGY
Volume 95, Issue 1, Pages 51-55Publisher
WILEY
DOI: 10.1113/expphysiol.2008.046334
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Funding
- USPHS NIH [R01 DA0017371, R21 HL091440, 5T32 GM008306]
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This brief review describes the role of neural and non-neural mechanisms during different phases of deoxycorticosterone acetate (DOCA)-salt hypertension. There are contradictory data for and against a role of the sympathetic nervous system and neurohumoral agents, including endothelin and vasopressin. Elucidating the factors responsible for DOCA-salt hypertension will be helpful in understanding the causes of hypertension resulting from hypervolaemia, hyperaldosteronism and high salt intake.
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