Journal
EMBO JOURNAL
Volume 32, Issue 11, Pages 1496-1498Publisher
NATURE PUBLISHING GROUP
DOI: 10.1038/emboj.2013.103
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Funding
- Intramural NIH HHS Funding Source: Medline
- Wellcome Trust Funding Source: Medline
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Mitochondria undergo dynamic fission and fusion events, and disruptions in this balance can lead to cellular dysfunction or death. New work by Henley and colleagues identifies a key role for the SUMO protease SENP3 and its target Drp1, a key mediator of mitochondrial fission, in ischaemia.
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