Journal
CLINICAL NUCLEAR MEDICINE
Volume 38, Issue 9, Pages 735-736Publisher
LIPPINCOTT WILLIAMS & WILKINS
DOI: 10.1097/RLU.0b013e31829b9bd9
Keywords
FDG; cerebral glucose metabolism; electroconvulsive therapy; PET
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We describe a patient with a complex neuropsychiatric disorder who presented a severe and diffuse cerebral glucose hypometabolism on F-18-FDG PET initially which, in the clinical setting, was suspicious of an advanced neurodegenerative disease. Further evaluation suggested a major depressive episode with agitation and poor response to medication. Electroconvulsive therapy (ECT) brought excellent results. A follow-up cerebral F-18-FDG PET was completely normal, thus illustrating the potential for complete recovery and normalization of brain metabolism in major depressive episode following ECT. It also shows the risk of false interpretation of brain PET in patients with depression.
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