Journal
CLINICAL ENDOCRINOLOGY
Volume 76, Issue 2, Pages 212-219Publisher
WILEY
DOI: 10.1111/j.1365-2265.2011.04159.x
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Funding
- Mayo Clinic CTSA [RR24150]
- Minnesota Obesity Center [DK50456]
- Mayo Clinic CR20
- [DK78646]
- [DK29953]
- [DK 67071]
- [HL-55502]
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Aims/hypothesis Individuals with impaired fasting glucose (IFG) are at increased risk of developing diabetes over the subsequent decade. However, there is uncertainty as to the mechanisms contributing to the development of diabetes. We sought to quantitate insulin secretion and action across the prediabetic range of fasting glucose.
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