Journal
CLINICAL AND EXPERIMENTAL PHARMACOLOGY AND PHYSIOLOGY
Volume 39, Issue 2, Pages 179-193Publisher
WILEY
DOI: 10.1111/j.1440-1681.2011.05641.x
Keywords
aldehydes; cardiac hypertrophy; diabetes; heart failure; ischaemia; lipid peroxidation products; mitochondria
Categories
Funding
- National Institutes of Health [R21HL098780, R01HL104129]
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1. The heart is a highly oxidative organ in which cardiomyocyte turnover is virtually absent, making it particularly vulnerable to accumulation of lipid peroxidation products (LPP) formed as a result of oxidative damage.
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