Related references
Note: Only part of the references are listed.Molecular mechanisms of angiotensin II-mediated mitochondrial dysfunction - Linking mitochondrial oxidative damage and vascular endothelial dysfunction
Abdulrahman K. Doughan et al.
CIRCULATION RESEARCH (2008)
Reduction of age-associated pathology in old mice by overexpression of catalase in mitochondria
Piper M. Treuting et al.
JOURNALS OF GERONTOLOGY SERIES A-BIOLOGICAL SCIENCES AND MEDICAL SCIENCES (2008)
DNA deletions and clonal mutations drive premature aging in mitochondrial mutator mice
Marc Vermulst et al.
NATURE GENETICS (2008)
Protective effect of long-term angiotensin II inhibition
Nidia Basso et al.
AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY (2007)
Small-molecule therapies for cardiac hypertrophy: moving beneath the cell surface
Timothy A. McKinsey et al.
NATURE REVIEWS DRUG DISCOVERY (2007)
Mitochondrial point mutations do not limit the natural lifespan of mice
Marc Vermulst et al.
NATURE GENETICS (2007)
Cardiac overexpression of antioxidant catalase attenuates aging-induced cardiomyocyte relaxation dysfunction
Jun Ren et al.
MECHANISMS OF AGEING AND DEVELOPMENT (2007)
Suppression of reactive oxygen species and neurodegeneration by the PGC-1 transcriptional coactivators
Julie St-Pierre et al.
CELL (2006)
Regulation of cardiac hypertrophy by intracellular signalling pathways
Joerg Heineke et al.
NATURE REVIEWS MOLECULAR CELL BIOLOGY (2006)
Overexpression of mitochondrial peroxiredoxin-3 prevents left ventricular remodeling and failure after myocardial infarction in mice
S Matsushima et al.
CIRCULATION (2006)
Mechanisms underlying variations in excitation-contraction coupling across the mouse left ventricular free wall
KW Dilly et al.
JOURNAL OF PHYSIOLOGY-LONDON (2006)
Redox-dependent transcriptional regulation
HJ Liu et al.
CIRCULATION RESEARCH (2005)
Angiotensin II-mediated phenotypic cardiomyocyte remodeling leads to age-dependent cardiac dysfunction and failure
AA Domenighetti et al.
HYPERTENSION (2005)
Extension of murine life span by overexpression of catalase targeted to mitochondria
SE Schriner et al.
SCIENCE (2005)
Quantification of random genomic mutations
JH Bielas et al.
NATURE METHODS (2005)
Mitochondria, oxidants, and aging
RS Balaban et al.
CELL (2005)
A practical approach to the echocardiographic evaluation of diastolic function
SJ Khouri et al.
JOURNAL OF THE AMERICAN SOCIETY OF ECHOCARDIOGRAPHY (2004)
TGF-β1 and angiotensin networking in cardiac remodeling
S Rosenkranz
CARDIOVASCULAR RESEARCH (2004)
Resistance to glomerulosclerosis in B6 mice disappears after menopause
F Zheng et al.
AMERICAN JOURNAL OF PATHOLOGY (2003)
Arterial and cardiac aging: Major shareholders in cardiovascular disease enterprises - Part III: Cellular and molecular clues to heart and arterial aging
EG Lakatta
CIRCULATION (2003)
Arterial and cardiac aging: Major shareholders in cardiovascular disease enterprises - Part II: The aging heart in health: Links to heart disease
EG Lakatta et al.
CIRCULATION (2003)
New concepts in diastolic dysfunction and diastolic heart failure: Part II - Causal mechanisms and treatment
MR Zile et al.
CIRCULATION (2002)
The antioxidant N-2-mercaptopropionyl glycine attenuates left ventricular hypertrophy in in vivo murine pressure-overload model
M Date et al.
JOURNAL OF THE AMERICAN COLLEGE OF CARDIOLOGY (2002)
The transcription factor GATA4 is activated by extracellular signal-regulated kinase 1-and 2-mediated phosphorylation of serine 105 in cardiomyocytes
QR Liang et al.
MOLECULAR AND CELLULAR BIOLOGY (2001)
Impaired cell shortening and relengthening with increased pacing frequency are intrinsic to the senescent mouse cardiomyocyte
CC Lim et al.
JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY (2000)
Abnormalities of calcium cycling in the hypertrophied and failing heart
SR Houser et al.
JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY (2000)