Related references
Note: Only part of the references are listed.Damage-induced ubiquitylation of human RNA polymerase II by the ubiquitin ligase Nedd4, but not Cockayne syndrome proteins or BRCA1
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被撤回的出版物: Cockayne syndrome A and B proteins differentially regulate recruitment of chromatin remodeling and repair factors to stalled RNA polymerase II in vivo (Retracted article. See vol. 81, pg. 5112, 2021)
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When transcription and repair meet:: a complex system
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Cockayne syndrome group B protein (CSB) plays a general role in chromatin maintenance and remodeling
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The Y-family DNA polymerase κ (pol κ) functions in mammalian nucleotide-excision repair
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CSA-dependent degradation of CSB by the ubiquitin-proteasome pathway establishes a link between complementation factors of the Cockayne syndrome
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RNA polymerase II blockage by cisplatin-damaged DNA - Stability and polyubiquitylation of stalled polymerase
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Enhanced DDB2 expression protects mice from carcinogenic effects of chronic UV-B irradiation
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Transcription-coupled repair: Impact on UV-induced mutagenesis in cultured rodent cells and mouse skin tumors
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Multiple mechanisms confining RNA polymerase II ubiquitylation to polymerases undergoing transcriptional arrest
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The UV-damaged DNA binding protein mediates efficient targeting of the nucleotide excision repair complex to UV-induced photo lesions
J Moser et al.
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Single-stranded breaks in DNA but not oxidative DNA base damages block transcriptional elongation by RNA polymerase II in HeLa cell nuclear extracts
SD Kathe et al.
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Fate of RNA polymerase II stalled at a cisplatin lesion
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Opinion - Transcription - guarding the genome by sensing DNA damage
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DNA damage stabilizes interaction of CSB with the transcription elongation machinery
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Transcription activities at 8-oxoG lesions in DNA
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Complete absence of Cockayne syndrome group B gene product gives rise to UV-sensitive syndrome but not Cockayne syndrome
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Effects of genomic context and chromatin structure on transcription-coupled and global genomic repair in mammalian cells
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The ubiquitin ligase activity in the DDB2 and CSA complexes is differentially regulated by the COP9 signalosome in response to DNA damage
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Chromosomal protein HMGN1 enhances the rate of DNA repair in chromatin
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Transcription-dependent degradation of topoisomerase I-DNA covalent complexes
SD Desai et al.
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A global DNA repair mechanism involving the Cockayne syndrome B (CSB) gene product can prevent the in vivo accumulation of endogenous, oxidative DNA base damage
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Ultraviolet-sensitive syndrome cells are defective in transcription-coupled repair of cyclobutane pyrimidine dimers
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Interfaces between the detection, signaling, and repair of DNA damage
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Sequential binding of UV DNA damage binding factor and degradation of the p48 subunit as early events after UV irradiation
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Repair of 8-oxoG is slower in endogenous nuclear genes than in mitochondrial DNA and is without strand bias
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A Rad26-Def1 complex coordinates repair and RNA pol II proteolysis in response to DNA damage
EC Woudstra et al.
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Translocation of Cockayne syndrome group A protein to the nuclear matrix: Possible relevance to transcription-coupled DNA repair
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Double-check probing of DNA bending and unwinding by XPA-RPA:: an architectural function in DNA repair
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Chromatin unfolding and activation by HMGN chromosomal proteins
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DMBA-induced toxic and mutagenic responses vary dramatically between NER-deficient Xpa, Xpc and Csb mice
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Sequential assembly of the nucleotide excision repair factors in vivo
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Structural basis of transcription:: RNA polymerase II at 2.8 Ångstrom resolution
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XAB2, a novel tetratricopeptide repeat protein involved in transcription-coupled DNA repair and transcription
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ATP-dependent chromatin remodeling by the Cockayne syndrome B DNA repair-transcription-coupling factor
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UV-induced inhibition of transcription involves repression of transcription initiation and phosphorylation of RNA polymerase II
DAP Rockx et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2000)