Journal
CELL METABOLISM
Volume 12, Issue 4, Pages 311-312Publisher
CELL PRESS
DOI: 10.1016/j.cmet.2010.09.007
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Despite its high prevalence, the mechanisms causing diabetic kidney disease remain poorly understood. In this issue of Cell Metabolism, Welsh et al. (2010) show that elimination of insulin receptors from the glomerular podocyte, a cell that is central to the pathogenesis of proteinuric renal diseases, recapitulates many features of human diabetic nephropathy.
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