Journal
CELL HOST & MICROBE
Volume 10, Issue 2, Pages 93-94Publisher
CELL PRESS
DOI: 10.1016/j.chom.2011.08.001
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The pathogen Candida albicans can occupy both the bloodstream and gastrointesintal (GI) tract, niches that differ in iron availability. Chen et al. report that a distinct transcription factor, Sef1, alters the conserved fungal iron regulatory paradigm. Sef1 is pivotal for bloodstream infection, but contributes to GI tract colonization as well.
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