Related references
Note: Only part of the references are listed.Pancreatic tumor cell metabolism: Focus on glycolysis and its connected metabolic pathways
Fabienne Guillaumond et al.
ARCHIVES OF BIOCHEMISTRY AND BIOPHYSICS (2014)
Molecular pathology of pancreatic cancer
Yuriko Saiki et al.
PATHOLOGY INTERNATIONAL (2014)
The role of FAK in tumor metabolism and therapy
Jianliang Zhang et al.
PHARMACOLOGY & THERAPEUTICS (2014)
Disruption of the protein interaction between FAK and IGF-1R inhibits melanoma tumor growth
Deniz A. Ucar et al.
CELL CYCLE (2012)
The Focal Adhesion Kinase Inhibitor GSK2256098: a Potent and Selective Inhibitor for the Treatment of Cancer
K. R. Auger et al.
EUROPEAN JOURNAL OF CANCER (2012)
Clinical Importance and Potential Use of Small Molecule Inhibitors of Focal Adhesion Kinase
Alexander Schultze et al.
ANTI-CANCER AGENTS IN MEDICINAL CHEMISTRY (2011)
Signaling Pathways in Pancreatic Cancer
Meir Preis et al.
CRITICAL REVIEWS IN EUKARYOTIC GENE EXPRESSION (2011)
Phosphorylation of Focal Adhesion Kinase at Tyr397 in Gastric Carcinomas and its Clinical Significance
I-Rue Lai et al.
AMERICAN JOURNAL OF PATHOLOGY (2010)
A novel small molecule inhibitor of FAK decreases growth of human pancreatic cancer
Steven N. Hochwald et al.
CELL CYCLE (2009)
An integrin αvβ3-c-Src oncogenic unit promotes anchorage-independence and tumor progression
Jay S. Desgrosellier et al.
NATURE MEDICINE (2009)
FAK and IGF-IR interact to provide survival signals in human pancreatic adenocarcinoma cells
Weiguo Liu et al.
CARCINOGENESIS (2008)
Increased expression of tissue transglutaminase in pancreatic ductal adenocarcinoma and its implications in drug resistance and metastasis
Amit Verma et al.
CANCER RESEARCH (2006)
Focal adhesion kinase, Rap1, and transcriptional induction of vascular endothelial growth factor
EA Sheta et al.
JOURNAL OF THE NATIONAL CANCER INSTITUTE (2000)