Journal
CELL
Volume 155, Issue 6, Pages 1216-1219Publisher
CELL PRESS
DOI: 10.1016/j.cell.2013.11.019
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Funding
- NIH [R37CA53370, RC1CA147961, R01CA163591, R01CA130893]
- Department of Defense [W81XWH-09-01-0394]
- Val Skinner Foundation
- New Jersey Commission for Cancer Research
- Rutgers Cancer Institute of New Jersey
- American Cancer Society [RSG TBG-119822]
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Mouse models for cancer are revealing novel cancer-promoting roles for autophagy. Autophagy promotes tumor growth by suppressing the p53 response, maintaining mitochondrial function, sustaining metabolic homeostasis and survival in stress, and preventing diversion of tumor progression to benign oncocytomas.
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