Journal
CELL
Volume 135, Issue 3, Pages 396-400Publisher
CELL PRESS
DOI: 10.1016/j.cell.2008.10.015
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Funding
- Howard Hughes Medical Institute Funding Source: Medline
- NIMH NIH HHS [1K23MH080954, K23 MH080954-05, K23 MH080954, 1R01 MH083565, R01 MH083565-02, R01 MH049428, R01 MH49428, R01 MH083565] Funding Source: Medline
- NINDS NIH HHS [R01 NS034661, R01 NS34661] Funding Source: Medline
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Genetic studies are refining our understanding of neurodevelopmental mechanisms in autism. Some autism-related mutations appear to disrupt genes regulated by neuronal activity, which are especially important in development of the postnatal nervous system. Gene replacement studies in mice indicate that the developmental window to ameliorate symptoms may be wider than previously anticipated.
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