Related references
Note: Only part of the references are listed.Specific residues of the cytoplasmic domains of cardiac inward rectifier potassium channels are effective antifibrillatory targets
Sami F. Noujaim et al.
FASEB JOURNAL (2010)
Prevention of Atrial Fibrillation Report From a National Heart, Lung, and Blood Institute Workshop
Emelia J. Benjamin et al.
CIRCULATION (2009)
The molecular basis of chloroquine block of the inward rectifier Kir2.1 channel
Aldo A. Rodriguez-Menchaca et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2008)
Phosphoinositide-mediated gating of inwardly rectifying K+ channels
Diomedes E. Logothetis et al.
PFLUGERS ARCHIV-EUROPEAN JOURNAL OF PHYSIOLOGY (2007)
Reduction of dispersion of repolarization and prolongation of postrepolarization refractoriness explain the antiarrhythmic effects of quinidine in a model of short QT syndrome
Peter Milberg et al.
JOURNAL OF CARDIOVASCULAR ELECTROPHYSIOLOGY (2007)
Up-regulation of the inward rectifier K+ current (I-K1) in the mouse heart accelerates and stabilizes rotors
Sami F. Noujaim et al.
JOURNAL OF PHYSIOLOGY-LONDON (2007)
Molecular physiology of cardiac repolarization
JM Nerbonne et al.
PHYSIOLOGICAL REVIEWS (2005)
A Kir2.1 gain-of-function mutation underlies familial atrial fibrillation
M Xia et al.
BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS (2005)
Electrocardiographic features in Andersen-Tawil syndrome patients with KCNJ2 mutations -: Characteristic T-U-wave patterns predict the KCNJ2 genotype
L Zhang et al.
CIRCULATION (2005)
A novel form of short QT syndrome (SQT3) is caused by a mutation in the KCNJ2 gene
SG Priori et al.
CIRCULATION RESEARCH (2005)
Cytoplasmic domain structures of Kir2.1 and Kir3.1 show sites for modulating gating and rectification
S Pegan et al.
NATURE NEUROSCIENCE (2005)
Further insights into the effect of quinidine in short QT syndrome caused by a mutation in HERG
C Wolpert et al.
JOURNAL OF CARDIOVASCULAR ELECTROPHYSIOLOGY (2005)
PRODRG:: a tool for high-throughput crystallography of protein-ligand complexes
AW Schüttelkopf et al.
ACTA CRYSTALLOGRAPHICA SECTION D-STRUCTURAL BIOLOGY (2004)
Loss-of-function mutations of the K+ channel gene KCNJ2 constitute a rare cause of long QT syndrome
H Fodstad et al.
JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY (2004)
Transgenic upregulation of IK1 in the mouse heart leads to multiple abnormalities of cardiac excitability
JD Li et al.
AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY (2004)
Blockade of the inward rectifying potassium current terminates ventricular fibrillation in the guinea pig heart
M Warren et al.
JOURNAL OF CARDIOVASCULAR ELECTROPHYSIOLOGY (2003)
Functional role of inward rectifier current in heart probed by Kir2.1 overexpression and dominant-negative suppression
J Miake et al.
JOURNAL OF CLINICAL INVESTIGATION (2003)
Functional and clinical characterization of KCNJ2 mutations associated with LQT7 (Andersen syndrome)
M Tristani-Firouzi et al.
JOURNAL OF CLINICAL INVESTIGATION (2002)
Rectification of the background potassium current - A determinant of rotor dynamics in ventricular fibrillation
FH Samie et al.
CIRCULATION RESEARCH (2001)
Molecular basis of functional voltage-gated K+ channel diversity in the mammalian myocardium
JM Nerbonne
JOURNAL OF PHYSIOLOGY-LONDON (2000)