Journal
CANCER PREVENTION RESEARCH
Volume 3, Issue 4, Pages 403-405Publisher
AMER ASSOC CANCER RESEARCH
DOI: 10.1158/1940-6207.CAPR-10-0042
Keywords
-
Categories
Funding
- NATIONAL CANCER INSTITUTE [R01CA140594, R01CA122794] Funding Source: NIH RePORTER
- NATIONAL INSTITUTE ON AGING [R01AG027757] Funding Source: NIH RePORTER
- NCI NIH HHS [R01 CA140594-01, R01 CA122794-04, R01 CA140594, R01 CA122794] Funding Source: Medline
- NIA NIH HHS [R01 AG027757, R01 AG027757-04] Funding Source: Medline
Ask authors/readers for more resources
This perspective on Deng et al. (beginning on p. 424 in this issue of the journal) examines the link between NF-kappa B and lung tumorigenesis. Experiments in genetically engineered mouse models of lung cancers are elucidating protumorigenic roles of NF-kappa B activation in lung cancer pathogenesis. Our growing understanding of the tumor-promoting NF-kappa B downstream effector pathways could lead to the development of novel approaches for lung cancer therapy and chemoprevention. Cancer Prev Res; 3(4); 403-5. (C)2010 AACR.
Authors
I am an author on this paper
Click your name to claim this paper and add it to your profile.
Reviews
Recommended
No Data Available
Share Paper
