Journal
APPETITE
Volume 49, Issue 2, Pages 353-367Publisher
ACADEMIC PRESS LTD- ELSEVIER SCIENCE LTD
DOI: 10.1016/j.appet.2007.01.015
Keywords
salt appetite; angiotensin; aldosterone; corticosterone; Richter
Categories
Funding
- NATIONAL HEART, LUNG, AND BLOOD INSTITUTE [R00HL096830] Funding Source: NIH RePORTER
- NATIONAL INSTITUTE OF DIABETES AND DIGESTIVE AND KIDNEY DISEASES [R29DK048061, R01DK066596, F32DK079710, R01DK048061] Funding Source: NIH RePORTER
- NHLBI NIH HHS [R00 HL096830] Funding Source: Medline
- NIDDK NIH HHS [R01 DK048061, R29 DK048061-06, DK66596, R29 DK048061, F32 DK079710-01, F32 DK079710, R01 DK066596, DK48061] Funding Source: Medline
Ask authors/readers for more resources
Nearly three-quarters of a century ago, Curt Richter removed the adrenal glands from rats and noted that the animal's vitality was dependent on its increased consumption of sodium chloride. In doing so, Richter revealed an innate behavioral mechanism that serves to maintain the hydromineral balance of an animal faced with sodium deficit. This experiment and others like it, led to the development of a field of research devoted to the investigation of salt appetite. The following is a discussion of how Richter's initial observations gave birth to an evolving field that incorporates multiple approaches to examine the drive to consume sodium. Published by Elsevier Ltd.
Authors
I am an author on this paper
Click your name to claim this paper and add it to your profile.
Reviews
Recommended
No Data Available