Journal
TRENDS IN MICROBIOLOGY
Volume 15, Issue 3, Pages 119-126Publisher
ELSEVIER SCIENCE LONDON
DOI: 10.1016/j.tim.2007.01.003
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Funding
- NATIONAL CANCER INSTITUTE [R01CA097093] Funding Source: NIH RePORTER
- NATIONAL INSTITUTE OF ALLERGY AND INFECTIOUS DISEASES [R01AI067952] Funding Source: NIH RePORTER
- NCI NIH HHS [CA097093] Funding Source: Medline
- NIAID NIH HHS [AI051686, AI067952] Funding Source: Medline
- Wellcome Trust [GR066559] Funding Source: Medline
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During infection, viruses attempt to hijack the cell while the host responds with various defense systems. Traditional defenses include the interferon response and apoptosis, but recent work suggests that this antiviral arsenal also includes the cellular DNA damage response machinery. The observation of interactions between viruses and cellular DNA repair proteins has not only uncovered new complexities of the virus-host interaction but is also reinforcing the view that viruses can reveal key regulators of cellular pathways through the proteins they target.
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