Journal
AUTOIMMUNITY
Volume 40, Issue 4, Pages 295-298Publisher
TAYLOR & FRANCIS LTD
DOI: 10.1080/08916930701358925
Keywords
apoptosis; C reactive protein; necrosis; rheumatoid arthritis; systemic lupus erythematosus
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C reactive protein (CRP) levels directly correlate with the disease activity of many inflammatory diseases, e. g. sepsis, infection, and various autoimmunopathies such as rheumatoid arthritis (RA) [1-7]. In contrast, insufficient CRP levels are implicated in the development of systemic lupus erythematosus (SLE) [8-16]. This article reports on the level-depended effects of CRP in various diseases. In detail we show that increased and decreased levels of CRP, as demonstrated in patients with RA and SLE, respectively can contribute to disease progression.
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