Journal
CANCER CELL
Volume 22, Issue 3, Pages 281-282Publisher
CELL PRESS
DOI: 10.1016/j.ccr.2012.08.019
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Funding
- NCI NIH HHS [R01 CA133557, P01 CA117969] Funding Source: Medline
- NIBIB NIH HHS [R01 EB010023] Funding Source: Medline
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Acinar-to-ductal metaplasia (ADM) results from pancreatic injury or KRAS activation, and is an early step in pancreatic cancer progression. In this issue of Cancer Cell, Ardito and colleagues and Navas and colleagues demonstrate that ADM- and KRAS-driven pancreatic cancer require EGFR signaling, revealing a mechanism for developmental reprogramming that primes tumorigenesis.
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