Journal
CANCER CELL
Volume 21, Issue 3, Pages 329-331Publisher
CELL PRESS
DOI: 10.1016/j.ccr.2012.03.001
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Funding
- NCI NIH HHS [R01 CA057683] Funding Source: Medline
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Recent genomic analyses of pediatric glioblastoma, a poorly understood tumor with dismal outcome, have identified mutations in histone H3 variants that affect critical amino acids in the tail. The findings extend discoveries of chromatin regulator inactivation and gain-of-function mutations by documenting alteration of a modifiable histone residue in human cancer.
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