4.7 Article

SPARC promoter hypermethylation in colorectal cancers can be reversed by 5-Aza-2′deoxycytidine to increase SPARC expression and improve therapy response

Related references

Note: Only part of the references are listed.
Article Biochemistry & Molecular Biology

A novel interaction between procaspase 8 and SPARC enhances apoptosis and potentiates chemotherapy sensitivity in colorectal cancers

Michelle J. Tang et al.

JOURNAL OF BIOLOGICAL CHEMISTRY (2007)

Article Oncology

Frequent inactivation of SPARC by promoter hypermethylation in colon cancers

Eungi Yang et al.

INTERNATIONAL JOURNAL OF CANCER (2007)

Article Medicine, Research & Experimental

Genome-wide expression analysis of therapy-resistant tumors reveals SPARC as a novel target for cancer therapy

IT Tai et al.

JOURNAL OF CLINICAL INVESTIGATION (2005)

Article Medicine, Research & Experimental

Aberrant CpG island hypermethylation of multiple genes in colorectal neoplasia

S Lee et al.

LABORATORY INVESTIGATION (2004)

Article Medicine, Research & Experimental

Enhanced growth of tumors in SPARC null mice is associated with changes the ECM

RA Brekken et al.

JOURNAL OF CLINICAL INVESTIGATION (2003)

Article Medicine, Research & Experimental

SPARC, a matricellular protein that functions in cellular differentiation and tissue response to injury

AD Bradshaw et al.

JOURNAL OF CLINICAL INVESTIGATION (2001)

Article Multidisciplinary Sciences

Inactivation of the apoptosis effector Apaf-1 in malignant melanoma

MS Soengas et al.

NATURE (2001)

Article Multidisciplinary Sciences

CpG methylation is maintained in human cancer cells lacking DNMT1

I Rhee et al.

NATURE (2000)