Journal
ENVIRONMENTAL EPIGENETICS
Volume 2, Issue 1, Pages -Publisher
OXFORD UNIV PRESS
DOI: 10.1093/eep/dvv008
Keywords
air pollution; preterm birth; epigenetics; DNA methylation; pregnancy
Categories
Funding
- NIEHS [R01ES020836, R01ES020268, R01ES021357, R01ES021733, R21ES020010, R21ES020984, R21ES024841]
- Harvard T.H. Chan-NIEHS Center for Environmental Health [P30ES00002]
- NIH-NIEHS [K23ES02224]
- NATIONAL INSTITUTE OF ENVIRONMENTAL HEALTH SCIENCES [R21ES024841, R21ES020010, R01ES020836, R21ES020984, R01ES021357, R01ES021733, K23ES022242, P30ES000002, R01ES020268] Funding Source: NIH RePORTER
Ask authors/readers for more resources
Preterm birth is a major cause of infant morbidity and mortality and a potential risk factor for adult chronic disease. With over 15 million infants born preterm worldwide each year, preterm birth poses a global health concern. There is a possible association between air pollution and preterm birth, though studies have been inconsistent, likely due to variation in study design. How air pollution induces health effects is uncertain; however, studies have repeatedly demonstrated the effects of air pollution on epigenetic modifications. More recent evidence suggests that epigenetics may, in turn, be linked to preterm birth. Discovery of environmentally modifiable epigenetic processes connected to preterm birth may help to identify women at risk of preterm birth, and ultimately lead to development of new preterm birth prevention measures.
Authors
I am an author on this paper
Click your name to claim this paper and add it to your profile.
Reviews
Recommended
No Data Available