Journal
BIRTH DEFECTS RESEARCH PART A-CLINICAL AND MOLECULAR TERATOLOGY
Volume 100, Issue 3, Pages 189-201Publisher
WILEY
DOI: 10.1002/bdra.23220
Keywords
invasive ventilation; infection; hyperoxia; lung; newborn
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Funding
- NIH [HL 085103]
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Exposure to hyperoxia, invasive mechanical ventilation, and systemic/local sepsis are important antecedents of postnatal inflammation in the pathogenesis of bronchopulmonary dysplasia (BPD). This review will summarize information obtained from animal (baboon, lamb/sheep, rat and mouse) models that pertain to the specific inflammatory agents and signaling molecules that predispose a premature infant to BPD. Birth Defects Research (Part A) 100:189-201, 2014. (c) 2014 Wiley Periodicals, Inc.
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