Journal
INFECTION AND IMMUNITY
Volume 68, Issue 7, Pages 4361-4362Publisher
AMER SOC MICROBIOLOGY
DOI: 10.1128/IAI.68.7.4361-4362.2000
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- PHS HHS [NIH 51873] Funding Source: Medline
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Mice deficient in phox (gp91(phox-/-)) or NOS2 (NOS2(-/-)) were infected with the agent of human granulocytic ehrlichiosis (HGE) to evaluate the importance of these pathways in the eradication of HGE bacteria, NOS2(-/-) mice had delayed clearance of the HGE agent in comparison to control or gp91(phox-/-) mice, suggesting that reactive nitrogen intermediates play a role in the early control of HGE.
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