Journal
AUTOIMMUNITY
Volume 42, Issue 7, Pages 574-579Publisher
TAYLOR & FRANCIS LTD
DOI: 10.1080/08916930903002461
Keywords
Angiogenesis; pathogenesis; psoriasis; autoimmune; skin
Categories
Funding
- National Institutes of Health [RO1 AR47901, P30 AR42687]
- NATIONAL INSTITUTE OF ARTHRITIS AND MUSCULOSKELETAL AND SKIN DISEASES [P30AR042687, R01AR047901] Funding Source: NIH RePORTER
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Psoriasis is a multifactorial skin disorder with known systemic consequences. While most recent studies point to a predominantly Th17-driven process, psoriasis also lies at the crossroads linking the pathways of angiogenesis and inflammation. Mediators such as VEGF and angiopoietin-2 play significant roles in the pathophysiology and may even account for the maintenance of the chronic inflammatory state. Targets in this shared pathway may offer alternative avenues for therapy.
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