Journal
PLANT CELL
Volume 14, Issue 2, Pages 301-319Publisher
AMER SOC PLANT BIOLOGISTS
DOI: 10.1105/tpc.010283
Keywords
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Funding
- NIGMS NIH HHS [R29 GM052456, R29-GM52456] Funding Source: Medline
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In Arabidopsis, SHY2 encodes 1AA3, a member of the auxin-induced Aux/1AA family. Gain-of-function mutations in SHY2/IAA3 cause enlarged cotyledons, short hypocotyls, and altered auxin-regulated root development. Here we show that the gain-of-function mutation shy2-2 decreases both the induction and repression of auxin-regulated genes, suggesting that SHY2/IAA3 acts as a negative regulator in auxin signaling. shy2-2 affects auxin induction of many previously characterized primary response genes, implying that it might repress primary auxin responses. In addition, shy2-2 also affects expression of multiple auxin-nonresponsive genes. Light regulates expression of SHY2/IAA3, suggesting a possible link between light and auxin response pathways.
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