Journal
INFECTION AND IMMUNITY
Volume 70, Issue 9, Pages 5299-5303Publisher
AMER SOC MICROBIOLOGY
DOI: 10.1128/IAI.70.9.5299-5303.2002
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Funding
- NINDS NIH HHS [NS31528] Funding Source: Medline
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GM(1)- and GD(1a)-like ganglioside mimicry in Campylobacter jejuni lipooligosaccharide (LOS) is considered to be involved in the pathogenesis of Campylobacter-induced Guillain-Barre syndrome (GBS). Compared with gastroenteritis-related isolates, GBS-related C. jejuni isolates were strongly associated with the expression of GDla-like mimicry. The presence of a few genes involved in LOS ganglioside mimicry, cst-II, cgtA, and cgtB, was also associated with GBS-related strains. GDla like epitope expression may be an important virulence phenotype associated with the risk of developing GBS following campylobacter infection.
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