Journal
TRENDS IN NEUROSCIENCES
Volume 26, Issue 2, Pages 81-89Publisher
ELSEVIER SCIENCE LONDON
DOI: 10.1016/S0166-2236(02)00040-1
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Ca2+ entry through the NMDA subtype of glutamate receptors has the power to determine whether neurons survive or die. Too much NMDA receptor activity is harmful to neurons - but so is too little. Is it a case of too much or too little Ca2+ influx causing cell death or do other factors, such as receptor location or receptor-associated proteins, play a role? Understanding the mechanisms behind this dichotomous signalling is an important area of molecular neuroscience with direct clinical implications.
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