Journal
NEWS IN PHYSIOLOGICAL SCIENCES
Volume 18, Issue -, Pages 89-94Publisher
AMER PHYSIOLOGICAL SOC
DOI: 10.1152/nips.01433.2002
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Mitochondria play a central part in cellular survival and apoptotic death. These processes are highly regulated by pro- and antiapoptotic Bcl-2 superfamily members. A key feature within apoptosis cascades is disruption of mitochondrial transmembrane potential and apoptogenic protein release, caused by opening of the permeability transition pore (PT). New data, however, indicate that mitochondrial apoptosis may occur without PT involvement.
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