Journal
ANTIMICROBIAL AGENTS AND CHEMOTHERAPY
Volume 47, Issue 6, Pages 2036-2039Publisher
AMER SOC MICROBIOLOGY
DOI: 10.1128/AAC.47.6.2036-2039.2003
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Funding
- NIAID NIH HHS [R01 AI040481, R01 AI40481-01A1] Funding Source: Medline
- PHS HHS [R03I 44999-01] Funding Source: Medline
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Four clinical U.S. glycopeptide intermediate resistant Staphylococcus aureus (GISA) isolates were resistant to Triton X-100-induced autolysis. Similar resistance was demonstrated in an isolate obtained after a single passage of a susceptible clinical isolate in low-level vancomycin. Strains with the vancomycin-induced Triton X-100 resistance phenotype produced active murein hydrolases but were resistant to lysis by murein hydrolases.
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