Journal
ARCHIVES OF GYNECOLOGY AND OBSTETRICS
Volume 288, Issue 3, Pages 501-505Publisher
SPRINGER HEIDELBERG
DOI: 10.1007/s00404-013-2774-3
Keywords
Apoptosis; Caspase-3; Preterm premature rupture of membranes; Preterm labor
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The present study was designed to examine apoptotic cell death via the caspase-dependent pathway in human fetal membranes. Amniotic membrane samples were collected from three groups of women: group 1, women with preterm premature rupture of fetal membranes (PPROM) after cesarean delivery (n = 10), group 2, women with preterm labor (PTL) with intact membranes after cesarean delivery (n = 9) and group 3, women with term labor and vaginal delivery after an uncomplicated pregnancy (controls) (n = 11). Active caspase-3 immunopositivity (ACPI) of the PPROM group was significantly higher than that of the control group (p < 0.05). ACPI was higher in the PTL with intact membranes group as compared to the control group; however, it did not reach statistical significance (p > 0.05). Active caspase-3 positivity is increased in the fetal membranes of those women with PPROM.
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