Journal
CURRENT OPINION IN GENETICS & DEVELOPMENT
Volume 14, Issue 3, Pages 292-300Publisher
CURRENT BIOLOGY LTD
DOI: 10.1016/j.gde.2004.04.001
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DNA damage that is not properly repaired during genomic replication is a major source of gross chromosomal rearrangements and sequence loss during cell proliferation. In higher eukaryotes such mutations increase the risk of cancer. Eukaryotic cells have multiple checkpoint responses activated by DNA damage and stalled replication forks. We focus here on fork-associated events that activate and respond to S-phase checkpoint kinases.
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