Journal
PFLUGERS ARCHIV-EUROPEAN JOURNAL OF PHYSIOLOGY
Volume 448, Issue 3, Pages 296-303Publisher
SPRINGER HEIDELBERG
DOI: 10.1007/s00424-004-1256-7
Keywords
nitric oxide; peroxynitrite; metallothionein; potassium channel; zinc; p38; apoptosis; Kv2.1
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Funding
- NINDS NIH HHS [R01 NS043277-09, R01 NS043277-10, R56 NS043277-06, R01 NS043277-07A2, R56 NS043277, R01 NS043277, R01 NS043277-02, R01 NS043277-08, NS43277] Funding Source: Medline
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Nitrosative stress has been implicated in a large number of neurological disorders. The molecular mechanisms underlying the neuronal injury associated with this stimulus, however, are not clearly understood. Emerging evidence suggests that the liberation of intracellular zinc as well as over-activation of potassium channels may be two important components of nitrosative stress-induced neuronal death.
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