Journal
JOURNAL OF CELL BIOLOGY
Volume 166, Issue 2, Pages 157-159Publisher
ROCKEFELLER UNIV PRESS
DOI: 10.1083/jcb.200406151
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Funding
- NCI NIH HHS [R01 CA090901, CA90901] Funding Source: Medline
- NHLBI NIH HHS [HL45100, P01 HL045100] Funding Source: Medline
- NIDCR NIH HHS [DE13079, P60 DE013079] Funding Source: Medline
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FAK and paxillin are important components in integrin-regulated signaling. New evidence suggests that these two proteins function in crosstalk between cell-matrix and cell-cell adhesions. Further, new insight suggests that under some conditions these proteins inhibit cell migration, in contrast to their established roles in several cell systems as positive regulators of cell adhesion and migration.
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