Journal
ANTIMICROBIAL AGENTS AND CHEMOTHERAPY
Volume 53, Issue 2, Pages 821-823Publisher
AMER SOC MICROBIOLOGY
DOI: 10.1128/AAC.00132-08
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Funding
- National Basic Research Program of China from the Ministry of Science and Technology [2005CB0523101]
- National Institutes of Health, U. S. Public Health Service [AI43312, AI57576]
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In the sequence upstream from qnrB (but not qnrA or qnrS) is a LexA binding site. qnrB was shown to be under SOS control by demonstrating that quinolone susceptibility decreased with increasing temperature in a strain with a recA441(Ts) allele, whereas qnrB expression increased in response to ciprofloxacin or mitomycin C in strains with an intact lexA gene.
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