Journal
PHYSIOLOGY
Volume 20, Issue -, Pages 382-389Publisher
AMER PHYSIOLOGICAL SOC
DOI: 10.1152/physiol.00029.2005
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Funding
- NIEHS NIH HHS [R01-ES-12187, R01-ES-04066] Funding Source: Medline
- NINDS NIH HHS [R01-NS-41043] Funding Source: Medline
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Biliverdin reductase (BVR) functions in cell signaling through three distinct tracks: a dual-specificity kinase that functions in the insulin receptor/MAPK pathways (25, 29, 51); a bzip-type transcription factor for ATF-2/CREB and HO-1 regulation (1, 25); and a reductase that catalyzes the conversion of biliverdin to bilirubin (27). These, together with the protein's primary and secondary features, intimately link BVR to the entire spectrum of cell-signaling cascades.
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