Journal
ANGIOLOGY
Volume 62, Issue 7, Pages 535-542Publisher
SAGE PUBLICATIONS INC
DOI: 10.1177/0003319710398010
Keywords
high-sensitive C-reactive protein; C3 complement; internal carotid artery restenosis; carotid endarterectomy
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Funding
- Ministry of Science, Republic of Serbia [41002, 173033]
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The role of inflammation is well established in the pathogenesis of atherosclerosis and an increased level of circulating inflammatory markers may predict the future risk of atherosclerosis progression and plaque rupture. C-reactive protein (CRP) identification by hypersensitive methods (high-sensitivity CRP [hsCRP]) has become a clinical and laboratory inflammation marker. Carotid endarterectomy (CEA) is a well-established procedure for carotid stenosis treatment which can reduce stroke rate. Internal carotid artery (ICA) restenosis reduction may be prevented by the anti-inflammatory effect of statins. This review considers the recent findings on the presence of hsCRP and C3 complement concentration and inflammatory plaque composition as well as their effects on ICA restenosis rate, following eversion CEA with emphasis on human studies.
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