Journal
IMMUNOLOGY AND CELL BIOLOGY
Volume 85, Issue 6, Pages 471-475Publisher
WILEY
DOI: 10.1038/sj.icb.7100097
Keywords
TLR4 signaling; NF-kappa B; ERK; cell cycle; apoptosis
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Following encounters with microbes, cellular activation programs that involve the control of proliferation and survival are initiated in follicular B cells either via the B-cell receptor in a specific antigen-defined manner, or through Toll-like receptors (TLRs) that recognize specific microbial products. This review summarizes and discusses recent findings that shed light on how the nuclear factor kappa B pathway controls and coordinates B-cell division and survival following TLR4 engagement.
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