Related references
Note: Only part of the references are listed.Glucosamine protects neonatal cardiomyocytes from ischemia-reperfusion injury via increased protein O-GlcNAc and increased mitochondrial Bcl-2
Voraratt Champattanachai et al.
AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY (2008)
Cross-talk between GlcNAcylation and phosphorylation: roles in insulin resistance and glucose toxicity
Ronald J. Copeland et al.
AMERICAN JOURNAL OF PHYSIOLOGY-ENDOCRINOLOGY AND METABOLISM (2008)
Increased protein O-GlcNAc modification inhibits inflammatory and neointimal responses to acute endoluminal arterial injury
Dongqi Xing et al.
AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY (2008)
Cardioprotection by N-acetylglucosamine linkage to cellular proteins
Steven P. Jones et al.
CIRCULATION (2008)
Increased O-GlcNAc levels during reperfusion lead to improved functional recovery and reduced calpain proteolysis
Jia Liu et al.
AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY (2007)
Glucosamine cardioprotection in perfused rat hearts associated with increased O-linked N-acetylglucosamine protein modification and altered p38 activation
Norbert Fulop et al.
AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY (2007)
The protective effects of PUGNAc on cardiac function after trauma-hemorrhage are mediated via increased protein O-GlcNAc levels
Luyun Zou et al.
SHOCK (2007)
Trauma patients in the intensive care unit: short- and long-term survival and predictors of 30-day mortality
A. Ulvik et al.
ACTA ANAESTHESIOLOGICA SCANDINAVICA (2007)
Role of protein O-linked N-acetyl-glucosamine in mediating cell function and survival in the cardiovascular system
Norbert Fulop et al.
CARDIOVASCULAR RESEARCH (2007)
Glucosamine protects neonatal cardiomyocytes from ischemia-reperfusion injury via increased protein-associated O-GlcNAc
Voraratt Champattanachai et al.
AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY (2007)
Improving outcomes in a regional trauma system: impact of a level III trauma center
Michael L. Barringer et al.
AMERICAN JOURNAL OF SURGERY (2006)
Glucosamine administration during resuscitation improves organ function after trauma hemorrhage
Shaolong Yang et al.
SHOCK (2006)
Yersinia YopJ acetylates and inhibits kinase activation by blocking phosphorylation
Sohini Mukherjee et al.
SCIENCE (2006)
Effect of intravenous amino acids on glutamine and protein kinetics in low-birth-weight preterm infants during the immediate neonatal period
MM Kadrofske et al.
AMERICAN JOURNAL OF PHYSIOLOGY-ENDOCRINOLOGY AND METABOLISM (2006)
Inhibition of O-GlcNAcase by PUGNAc is dependent upon the oxime stereochemistry
M Perreira et al.
BIOORGANIC & MEDICINAL CHEMISTRY (2006)
Increased hexosamine biosynthesis and protein O-GlcNAc levels associated with myocardial protection against calcium paradox and ischemia
J Liu et al.
JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY (2006)
O-GlcNAc cycling: How a single sugar post-translational modification is changing the way we think about signaling networks
C Slawson et al.
JOURNAL OF CELLULAR BIOCHEMISTRY (2006)
O-GlcNAcase uses substrate-assisted catalysis -: Kinetic analysis and development of highly selective mechanism-inspired inhibitors
MS Macauley et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2005)
Glucosamine induces cell-cycle arrest and hypertrophy of mesangial cells: implication of gangliosides
E Masson et al.
BIOCHEMICAL JOURNAL (2005)
Pathophysiology of polytrauma
M Keel et al.
INJURY-INTERNATIONAL JOURNAL OF THE CARE OF THE INJURED (2005)
Combat casualty care research: From bench to the battlefield
HB Alam et al.
WORLD JOURNAL OF SURGERY (2005)
Gender differences in acute response to trauma-hemorrhage
MA Choudhry et al.
SHOCK (2005)
Adult rat cardiomyocytes exhibit capacitative calcium entry
DL Hunton et al.
AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY (2004)
Dynamic O-GlcNAc modification of nucleocytoplasmic proteins in response to stress -: A survival response of mammalian cells
NE Zachara et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2004)
O-GlcNAc a sensor of cellular state:: the role of nucleocytoplasmic glycosylation in modulating cellular function in response to nutrition and stress
NE Zachara et al.
BIOCHIMICA ET BIOPHYSICA ACTA-GENERAL SUBJECTS (2004)
Mechanism of cardiac depression after trauma-hemorrhage: increased cardiomyocyte IL-6 and effect of sex steroids on IL-6 regulation and cardiac function
SL Yang et al.
AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY (2004)
O-GIcNAc modification is an endogenous inhibitor of the proteasome
FX Zhang et al.
CELL (2003)
O-GlcNAc: a regulatory post-translational modification
L Wells et al.
BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS (2003)
Hyperglycemia inhibits capacitative calcium entry and hypertrophy in neonatal cardiomyocytes
Y Pang et al.
DIABETES (2002)
Capacitative calcium entry contributes to nuclear factor of activated T-cells nuclear translocation and hypertrophy in cardiomyocytes
DL Hunton et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2002)
Flux through the hexosamine pathway is a determinant of nuclear factor κB-dependent promoter activation
LR James et al.
DIABETES (2002)
Molecular mechanisms in the early phase of hemorrhagic shock
C Hierholzer et al.
LANGENBECKS ARCHIVES OF SURGERY (2001)
Gender differences in the inflammatory response and survival following haemorrhage and subsequent sepsis
MD Diodato et al.
CYTOKINE (2001)
Calpain inhibitor I reduces the activation of nuclear factor-kappa B and organ injury/dysfunction in hemorrhagic shock
MC McDonald et al.
FASEB JOURNAL (2001)
Effect of gender and sex hormones on immune responses following shock
MK Angele et al.
SHOCK (2000)
The aromatase inhibitor, 4-hydroxyandrostenedione, restores immune responses following trauma-hemorrhage in males and decreases mortality from subsequent sepsis
CP Schneider et al.
SHOCK (2000)
Share Paper
