Related references
Note: Only part of the references are listed.Inhibition of mitochondrial permeability transition pore opening by ischemic preconditioning is probably mediated by reduction of oxidative stress rather than mitochondrial protein phosphorylation
Samantha J. Clarke et al.
CIRCULATION RESEARCH (2008)
Polyol pathway mediates iron-induced oxidative injury in ischemic-reperfused rat heart
Wai Ho Tang et al.
FREE RADICAL BIOLOGY AND MEDICINE (2008)
Identification by a differential proteomic approach of the induced stress and redox proteins by resveratrol in the normal and diabetic rat heart
D. H. W. Dekkers et al.
JOURNAL OF CELLULAR AND MOLECULAR MEDICINE (2008)
Urocortin prevents mitochondrial permeability transition in response to reperfusion injury indirectly by reducing oxidative stress
Paul A. Townsend et al.
AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY (2007)
Deletion of aldose reductase leads to protection against cerebral ischemic injury
Amy C. Y. Lo et al.
JOURNAL OF CEREBRAL BLOOD FLOW AND METABOLISM (2007)
Design and synthesis of compounds that extend yeast replicative lifespan
Hongying Yang et al.
AGING CELL (2007)
The red wine antioxidant resveratrol prevents cardiomyocyte injury following ischemia-reperfusion via multiple sites and mechanisms
Siew Simg C. Goh et al.
ANTIOXIDANTS & REDOX SIGNALING (2007)
Resveratrol improves mitochondrial function and protects against metabolic disease by activating SIRT1 and PGC-1α
Marie Lagouge et al.
CELL (2006)
被撤回的出版物: Differential proteomic profiling to study the mechanism of cardiac pharmacological preconditioning by resveratrol (Retracted article. See vol. 16, pg. 2548, 2012)
Karel Bezstarosti et al.
JOURNAL OF CELLULAR AND MOLECULAR MEDICINE (2006)
Aldose reductase inhibitors improve myocardial reperfusion injury in mice by a dual mechanism
Kazumi Iwata et al.
JOURNAL OF PHARMACOLOGICAL SCIENCES (2006)
Aldose reductase-deficient mice are protected from delayed motor nerve conduction velocity, increased c-Jun NH2-terminal kinase activation, depletion of reduced glutathione, increased superoxide accumulation, and DNA damage
Eric C. M. Ho et al.
DIABETES (2006)
Increased sorbitol pathway activity generates oxidative stress in tissue sites for diabetic complications
IG Obrosova
ANTIOXIDANTS & REDOX SIGNALING (2005)
Human aldose reductase expression accelerates diabetic atherosclerosis in transgenic mice
RK Vikramadithyan et al.
JOURNAL OF CLINICAL INVESTIGATION (2005)
Nitric oxide donors protect murine myocardium against infarction via modulation of mitochondrial permeability transition
GW Wang et al.
AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY (2005)
Aldose reductase pathway mediates JAK-STAT signaling: a novel axis in myocardial ischemic injury
YYC Hwang et al.
FASEB JOURNAL (2005)
Decrease in mitochondrial complex I activity in ischemic/reperfused rat heart - Involvement of reactive oxygen species and cardiolipin
G Paradies et al.
CIRCULATION RESEARCH (2004)
Oxidative stress in the pathogenesis of diabetic neuropathy
AM Vincent et al.
ENDOCRINE REVIEWS (2004)
Central role for aldose reductase pathway in myocardial ischemic injury
YYC Hwang et al.
FASEB JOURNAL (2004)
Blockade of electron transport during ischemia protects cardiac mitochondria
EJ Lesnefsky et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2004)
Sorbitol dehydrogenase: a novel target for adjunctive protection of ischemic myocardium
YYC Hwang et al.
FASEB JOURNAL (2003)
Second window of ischemic preconditioning regulates mitochondrial permeability transition pore by enhancing Bcl-2 expression
KG Rajesh et al.
CARDIOVASCULAR RESEARCH (2003)
Contribution of polyol pathway to diabetes-induced oxidative stress
SSM Chung et al.
JOURNAL OF THE AMERICAN SOCIETY OF NEPHROLOGY (2003)
Aldose reductase inhibitor fidarestat prevents retinal oxidative stress and vascular endothelial growth factor overexpression in streptozotocin-diabetic rats
IG Obrosova et al.
DIABETES (2003)
Decreased complex III activity in mitochondria isolated from rat heart subjected to ischemia and reperfusion: role of reactive oxygen species and cardiolipin
G Petrosillo et al.
FASEB JOURNAL (2003)
Inhibiting mitochondrial permeability transition pore opening: a new paradigm for myocardial preconditioning?
DJ Hausenloy et al.
CARDIOVASCULAR RESEARCH (2002)
Aldose reductase is an obligatory mediator of the late phase of ischemic preconditioning
K Shinmura et al.
CIRCULATION RESEARCH (2002)
Pharmacological preconditioning with resveratrol: an insight with iNOS knockout mice
G Imamura et al.
AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY (2002)
Glycerophosphate-dependent hydrogen peroxide production by brown adipose tissue mitochondria and its activation by ferricyanide
Z Drahota et al.
JOURNAL OF BIOENERGETICS AND BIOMEMBRANES (2002)
Hyperglycemia increases endothelial superoxide that impairs smooth muscle cell Na+-K+-ATPase activity
S Gupta et al.
AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY (2002)
Protection of ischemic hearts by high glucose is mediated, in part, by GLUT-4
R Ramasamy et al.
AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY (2001)
Mitochondrial DNA damage and dysfunction associated with oxidative stress in failing hearts after myocardial infarction
T Ide et al.
CIRCULATION RESEARCH (2001)
A mitochondrial perspective on cell death
P Bernardi et al.
TRENDS IN BIOCHEMICAL SCIENCES (2001)
Opening of the mitochondrial permeability transition pore causes depletion of mitochondrial and cytosolic NAD+ and is a causative event in the death of myocytes in postischemic reperfusion of the heart
F Di Lisa et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2001)
Aldose reductase inhibition alone or combined with an adenosine A3 agonist reduces ischemic myocardial injury
WR Tracey et al.
AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY (2000)
Protection of hearts from reperfusion injury by propofol is associated with inhibition of the mitochondrial permeability transition
SA Javadov et al.
CARDIOVASCULAR RESEARCH (2000)
Share Paper
