4.6 Article

Deletion of Rab GAP AS160 modifies glucose uptake and GLUT4 translocation in primary skeletal muscles and adipocytes and impairs glucose homeostasis

Related references

Note: Only part of the references are listed.
Article Endocrinology & Metabolism

Insulin stimulates fusion, but not tethering, of GLUT4 vesicles in skeletal muscle of HA-GLUT4-GFP transgenic mice

Vladimir A. Lizunov et al.

AMERICAN JOURNAL OF PHYSIOLOGY-ENDOCRINOLOGY AND METABOLISM (2012)

Article Endocrinology & Metabolism

Thr649Ala-AS160 knock-in mutation does not impair contraction/AICAR-induced glucose transport in mouse muscle

Serge Ducommun et al.

AMERICAN JOURNAL OF PHYSIOLOGY-ENDOCRINOLOGY AND METABOLISM (2012)

Article Endocrinology & Metabolism

The Rab-GTPase-activating protein TBC1D1 regulates skeletal muscle glucose metabolism

Ferenc Szekeres et al.

AMERICAN JOURNAL OF PHYSIOLOGY-ENDOCRINOLOGY AND METABOLISM (2012)

Article Pharmacology & Pharmacy

Carbohydrate- and lipid-enriched meals acutely disrupt glycemic homeostasis by inducing transient insulin resistance in rats

Raquel Saldanha Campello et al.

CANADIAN JOURNAL OF PHYSIOLOGY AND PHARMACOLOGY (2012)

Article Cell Biology

Immunofluorescent localization of the Rab-GAP protein TBC1D4 (AS160) in mouse kidney

Natascha Lier et al.

HISTOCHEMISTRY AND CELL BIOLOGY (2012)

Article Biochemistry & Molecular Biology

AS160 Phosphotyrosine-binding Domain Constructs Inhibit Insulin-stimulated GLUT4 Vesicle Fusion with the Plasma Membrane

Francoise Koumanov et al.

JOURNAL OF BIOLOGICAL CHEMISTRY (2011)

Article Cell Biology

Exocyst function is regulated by effector phosphorylation

Xiao-Wei Chen et al.

NATURE CELL BIOLOGY (2011)

Article Cell Biology

Mapping Insulin/GLUT4 Circuitry

Alexander F. Rowland et al.

TRAFFIC (2011)

Article Endocrinology & Metabolism

Prenatal androgen exposure programs metabolic dysfunction in female mice

Alison V. Roland et al.

JOURNAL OF ENDOCRINOLOGY (2010)

Article Biochemistry & Molecular Biology

Insulin-stimulated Phosphorylation of the Rab GTPase-activating Protein TBC1D1 Regulates GLUT4 Translocation

Grantley R. Peck et al.

JOURNAL OF BIOLOGICAL CHEMISTRY (2009)

Article Cell Biology

Muscle cells engage Rab8A and myosin Vb in insulin-dependent GLUT4 translocation

Shuhei Ishikura et al.

AMERICAN JOURNAL OF PHYSIOLOGY-CELL PHYSIOLOGY (2008)

Review Biochemistry & Molecular Biology

Insulin action on glucose transporters through molecular switches, tracks and tethers

Hilal Zaid et al.

BIOCHEMICAL JOURNAL (2008)

Article Biochemistry & Molecular Biology

Complementary regulation of TBC1D1 and AS160 by growth factors, insulin and AMPK activators

Shuai Chen et al.

BIOCHEMICAL JOURNAL (2008)

Article Biochemistry & Molecular Biology

Discovery of TBC1D1 as an insulin-, AICAR-, and contraction-stimulated signaling nexus in mouse skeletal muscle

Eric B. Taylor et al.

JOURNAL OF BIOLOGICAL CHEMISTRY (2008)

Article Genetics & Heredity

Tbc1d1 mutation in lean mouse strain confers leanness and protects from diet-induced obesity

Alexandra Chadt et al.

NATURE GENETICS (2008)

Article Biochemistry & Molecular Biology

Substrate specificity and effect on GLUT4 translocation of the Rab GTPase-activating protein Tbc1d1

William G. Roach et al.

BIOCHEMICAL JOURNAL (2007)

Article Biochemistry & Molecular Biology

AS160 regulates insulin- and contraction-stimulated glucose uptake in mouse skeletal muscle

Henning F. Kramer et al.

JOURNAL OF BIOLOGICAL CHEMISTRY (2006)

Article Multidisciplinary Sciences

Smooth muscle of telokin-deficient mice exhibits increased sensitivity to Ca2+ and decreased cGMP-induced relaxation

AS Khromov et al.

PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2006)

Article Biochemistry & Molecular Biology

Characterization of the role of the Rab GTPase-activating protein AS160 in insulin-regulated GLUT4 trafficking

M Larance et al.

JOURNAL OF BIOLOGICAL CHEMISTRY (2005)

Article Biochemistry & Molecular Biology

Insulin hypersensitivity and resistance to streptozotocin-induced diabetes in mice lacking PTEN in adipose tissue

C Kurlawalla-Martinez et al.

MOLECULAR AND CELLULAR BIOLOGY (2005)

Article Physiology

GLUT4 activation: thoughts on possible mechanisms

LM Furtado et al.

ACTA PHYSIOLOGICA SCANDINAVICA (2003)

Article Biochemistry & Molecular Biology

Insulin-stimulated phosphorylation of a Rab GTPase-activating protein regulates GLUT4 translocation

H Sano et al.

JOURNAL OF BIOLOGICAL CHEMISTRY (2003)

Article Endocrinology & Metabolism

Mice lacking insulin receptor substrate 4 exhibit mild defects in growth, reproduction, and glucose homeostasis

VR Fantin et al.

AMERICAN JOURNAL OF PHYSIOLOGY-ENDOCRINOLOGY AND METABOLISM (2000)