Journal
AIDS
Volume 27, Issue 11, Pages 1829-1832Publisher
LIPPINCOTT WILLIAMS & WILKINS
DOI: 10.1097/QAD.0b013e328363089f
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Funding
- NIAID NIH HHS [R01 AI058740, T32 AI007343] Funding Source: Medline
- CSRD VA [I01 CX000821] Funding Source: Medline
- BLRD VA [I01 BX001241, I01 BX000207] Funding Source: Medline
- VA [5I01BX000207-06, 600959] Funding Source: Federal RePORTER
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GB virus C (GBV-C), a pan-lymphotropic flavivirus capable of persistent infection, is associated with prolonged survival and reduced T-cell activation in HIV-infected patients. GBV-C was associated with reduced CD56brt/CD16 S natural killer cell and monocyte activation, and a trend toward reduced B-cell activation by measuring cell surface activation markers or HIV entry coreceptors. The GBV-C association was independent of HIV viral load. Thus, GBV-C may influence non-T-cell immune activation in individuals with HIV infection.
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